Call (754) 547-2350Neuropathy evaluation in Lighthouse Point, FL
Physician-supervised neuropathy care

Root-Cause Neuropathy Treatment in Lighthouse Point, FL

We establish what is damaging your nerves before we treat it. The Rebuild Neuropathy Protocol™ matches a combination of six clinical modalities to your specific root cause rather than selling one package to everyone.

Why this is different

Diagnosis first, then a treatment plan

Neuropathy is a symptom pattern, not a diagnosis. The same burning, numbness and night pain can come from uncontrolled blood sugar, a compressed nerve root, a B12 deficiency, an untreated thyroid condition, or residual damage from chemotherapy. Those causes do not respond to the same treatment, yet a flat-fee package of fixed sessions is often quoted before anyone has established which one is driving the symptoms. We work in the other order: establish the cause, then build the protocol around it.

The workup comes before the quoteYour first visit is diagnostic. We review your history, current medications, recent labs including A1c and B12, and any prior nerve conduction study, then examine the affected territories with monofilament, vibration and reflex testing. What we find determines what we recommend. Pricing is discussed once there is something specific to price.
Six modalities, combined on purposeThe clinic uses Class IV laser, shockwave, IV nerve-support infusions, hormonal correction, metabolic optimization and mechanical decompression. Few patients need all six. A patient with poor glycemic control and a patient with an entrapped nerve root receive genuinely different combinations, because they have genuinely different problems.
We tell you where the ceiling isNerve tissue that has degenerated completely does not come back. If your nerve conduction study shows absent potentials rather than reduced amplitudes, we will say so at consultation rather than after you have paid for a course of treatment. Candidacy screening is part of the protocol, not an afterthought.

Start with the cause

Neuropathy is not one condition

Five root causes present with overlapping symptoms and respond to completely different treatment. Identifying which one you have is the first appointment, not an afterthought.

Root causeDiabetic NeuropathyDiabetic neuropathy is nerve damage caused by prolonged exposure of peripheral nerves to elevated blood glucose. It affects the longest nerves first, which is why symptoms usually start in the toes and move up the feet over months or years. The damage involves both the sensory fibers themselves and the small vessels that feed them, the vasa nervorum.Read more →
Root causePeripheral NeuropathyPeripheral neuropathy is damage to the nerves carrying signals between the spinal cord and the limbs. When testing does not identify diabetes, compression, chemotherapy or another clear driver, the case is often labeled idiopathic. A correctable nutritional or hormonal deficiency turns up in a meaningful share of those cases once someone runs the labs.Read more →
Root causeCompressive NeuropathyCompressive neuropathy is nerve dysfunction caused by mechanical pressure on a nerve root or a peripheral nerve rather than by a systemic metabolic process. The pressure may come from a herniated disc, a narrowed spinal canal, or fibrotic tissue that has formed around a nerve after chronic inflammation or injury. Because a specific nerve is affected, the symptoms follow that nerve's territory instead of appearing symmetrically in both feet.Read more →
Root causeMetabolic NeuropathyMetabolic neuropathy is nerve damage driven by a systemic metabolic or endocrine disturbance rather than by direct mechanical injury. Thyroid deficiency, low testosterone, estrogen deficiency, insulin resistance and prediabetes all alter the chemical environment peripheral nerves depend on. The resulting symptoms look much like other distal neuropathies, which is why the driver is only found when it is specifically tested for.Read more →
Root causeChemotherapy-Induced NeuropathyChemotherapy-induced peripheral neuropathy is nerve damage caused by neurotoxic cancer treatment rather than by a metabolic or mechanical process. Platinum agents damage the sensory neurons of the dorsal root ganglion, while taxanes and vinca alkaloids disrupt the microtubule transport that keeps long axons alive. The result is usually a symmetric glove-and-stocking pattern in the hands and feet that tracks with cumulative dose.Read more →

The Rebuild Neuropathy Protocol™

The Rebuild Neuropathy Protocol™

The protocol is a sequence, not a package. It begins with root-cause diagnosis, moves to a modality combination matched to what that diagnosis found, and continues through an initial treatment course that most patients complete over eight to twelve weeks. Maintenance is typically quarterly after that, adjusted to how the nerves respond.

  1. 01
    Root-cause diagnosis
    The first visit establishes what is damaging the nerve, not merely that something is. We review history, the full medication and supplement list, recent labs, and any prior nerve conduction study or EMG, then examine the affected territories with monofilament, vibration and reflex testing. Where the picture is incomplete, we order what is missing: A1c, B12, a thyroid panel, hormone levels, or imaging when root compression is suspected. Nothing is scheduled until there is a working cause.
  2. 02
    Modality matching
    Six modalities are available and most patients need three or four. Compressive findings bring in mechanical decompression with the Antalgic-Trac®. Poor glycemic control and an inflammatory metabolic picture bring in metabolic optimization and IV nerve support. Documented thyroid or sex hormone deficiency brings in hormonal correction. Your clinician explains which modalities were selected and, just as usefully, which were left out and why.
  3. 03
    Active treatment
    The initial course typically runs eight to twelve weeks of scheduled in-clinic sessions. Laser and shockwave are applied over the affected nerve territories, infusions are given in clinic, and the metabolic and hormonal components are managed alongside them under physician supervision. Progress is reassessed during the course using the same measures taken at baseline, so change is tracked rather than assumed. If a component is not contributing, it comes out of the plan.
  4. 04
    Maintenance
    Peripheral nerves regenerate slowly, and the metabolic conditions that damaged them usually persist. After the initial course, most patients move to a maintenance schedule that is typically quarterly, adjusted to how the nerves have responded. Maintenance is lighter than the initial protocol. Its purpose is to hold the ground gained rather than repeat the full course.

Six modalities

Combined to match the cause

No single modality treats every neuropathy. The combination is selected after the evaluation, and it differs between patients who arrive with the same symptoms.

What neuropathy actually is

Peripheral neuropathy is damage to the nerves carrying signals between the spinal cord and the hands, feet and organs. Patients usually describe burning, pins and needles, numbness that starts in the toes, or a loss of balance in the dark. Symptoms tend to follow a length-dependent pattern, which is why the feet are affected before the hands. Left unaddressed, sensory loss raises the risk of unnoticed foot wounds, particularly in patients with diabetes.

Who tends to do well here

Response is best when the underlying driver is still correctable. Diabetic peripheral neuropathy caught before advanced axonal loss, compressive neuropathy with a reversible mechanical component, and nutritional or hormonal neuropathy where the deficiency can be normalized are the situations this approach has the most to work with. Patients with decades of uncontrolled diabetes or long-term chemotherapy exposure may carry permanent structural damage that limits the ceiling of functional recovery. We screen for that before starting, not after.

What we will tell you plainly

Where this protocol reaches its limit

Conservative regenerative treatment addresses the functional spectrum of neuropathy, which means there has to be surviving nerve tissue to work with. Patients with advanced axonal degeneration from decades of uncontrolled diabetes or long-term chemotherapy may have permanent structural damage that limits the ceiling of functional recovery. End-stage neuropathy with complete loss of sensory or motor function, confirmed on nerve conduction studies showing absent potentials rather than reduced amplitudes, is irreversible structural damage, and this protocol does not reverse complete denervation. We would rather tell you that at consultation than take you through a course of treatment that cannot help.

Find out what is actually causing the nerve damage.

Evaluation happens in clinic at 3320 N Federal Hwy, Lighthouse Point. Pricing is quoted at consultation.